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Sinoatrial exit block type II. Intermittent sinus pauses with the
pause interval equals to the exact multiple of the preceding PP interval.
In this case, the pause is 2x of PP.
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Brugada ECG: J point elevation with coved-type ST elevation in V1-V2.
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Hypertrophic cardiomyopathy: high voltage in left-sided leads with ST depression and
T wave inversion.
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Long QT
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Old MI; Noted with Q waves in inferior and anterior leads.
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Left: sinus rhythm with pre-excitation (delta wave; slurred QRS upstroke) = WPW EKG.
Right: AF with extremely fast conduction via accessory pathway or AF with WPW.
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Arrhythmogenic right ventricular dysplasia (ARVD), a disease of right ventricule.
Characteristic ECG findings include Epsilon wave (delay in depolarization) and
T wave inversion in V1-V4.
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Early repolarization: J point elevation concaved downward ST elevation.
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Dual chamber pacemaker with failure to sense and pace of V lead.
Given the timing of 1wk after implantation, lead dislodgement is most likely.
s/p VVI 10years+
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Single chamber pacemaker with failure to sense and pace of V lead.
Given the timing of 10years after implantation, lead fracture, insulation break, or
battery depletion are possible. In this case, lead impedance was 1800 Ohm, so lead fracture
was the cause.
DDD 60/120 AVD 170.
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A and V leads switch in the header. Ventricular pacing is being inhibited by P wave (middle tracing),
ruling out lead dislodgement (there needs to be a lead in atrium).
Inhibition of V pacing is intermittent due to higher sensitivity in A channel.
50F with complete heart block s/p DDD 8 years ago, presents with syncope.
Pacemaker parameters are within normal ranges. Holter recording is as shown.
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Intermittent failure to pace the ventricle is noted. Oversensing is suspected.
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Normal pacemaker function. DDI 60, AVD 200.
VVI 50.
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Normal pacemaker function. Rhythm = sinus rhythm with AV Wenckebach.
V pacing came on the dropped beats.
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Pacemaker mediated tachycardia; triggered by a PVC with retrograde P, followed
by an non-capture A pacing with long AV delay, allowing the following retrograde
VA conduction to capture the atrium and initiate PMT.
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Sinus tachycardia with pacemaker Wenckebach.
TARP = 200+120, UTR = 110 bpm = 546 ms, 2:1 block @ 320 ms or 188 bpm,
WKB 320-546 ms or 188 to 110 bpm.
DDD 70/110
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Safety pacing triggered by timely PVCs. Noted with short AV delays.
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Managed ventricular pacing: AAI mode switches to DDD with short AV delay after
AP without native ventricular beat.
CRT non-responder. What is the next appropriate management?
more info
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Atrial flutter with intermittent biventricular pacing. Noted with beats of fusion
and native LBBB.
65M s/p CRT, in ICU with pneumonia and respiratory failure. The device is programmed
DDDR, 60-125 bpm.
The most likely explanation for the below tracing is:
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Sinus tachycardia above upper rate limit; hence no biventricular pacing.
1hr after DDD implantation.
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The atrial lead has dislodged into the ventricle. Ventricular captures are seen
by the first spike (from atrial lead) followed by a functional non-capture from the second spike (from
ventricular lead).
1wk post CRT
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Normal CRT function with VV delay of 40ms.
VVIR; post chest radiation
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Pacemaker failure. Radiation may cause deterioration of lead and pacemaker
circuit. ECG show asynchronous pacing. Urgent generator change is needed.
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Atrial undersensing. Some of A pacing spikes occur at the time of native QRS causing
safety pacing.