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1st degree AV block. In a severe case (PR>330ms), symptoms similar to pacemaker syndrome
may occur.
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Intermittent complete heart block. 2Ps without any QRS. Underlying
rhythm is sinus rhythm with RBBB and LAFB.
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Sinus pause
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AF with conversion pause
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Atrial Fibrillation: irregularly irregular rhythm. Noted with fibrillatory waves in V1.
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Polymorphic VT with long QT or Torsade de Pointes.
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Ischemic VT in a patient with history of inferior wall MI.
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Monomorphic VT. VA dissociation is clearly seen.
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Brugada ECG: J point elevation with coved-type ST elevation in V1-V2.
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Hypertrophic cardiomyopathy: high voltage in left-sided leads with ST depression and
T wave inversion.
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Long QT
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Old MI; Noted with Q waves in inferior and anterior leads.


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Arrhythmogenic right ventricular dysplasia (ARVD), a disease of right ventricule.
Characteristic ECG findings include Epsilon wave (delay in depolarization) and
T wave inversion in V1-V4.
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Early repolarization: J point elevation concaved downward ST elevation.
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Short QT
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A and V leads switch in the header. Ventricular pacing is being inhibited by P wave (middle tracing),
ruling out lead dislodgement (there needs to be a lead in atrium).
Inhibition of V pacing is intermittent due to higher sensitivity in A channel.
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Normal pacemaker function. DDI 60, AVD 200.
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Normal pacemaker function. Rhythm = sinus rhythm with AV Wenckebach.
V pacing came on the dropped beats.
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Pacemaker mediated tachycardia; triggered by a PVC with retrograde P, followed
by an non-capture A pacing with long AV delay, allowing the following retrograde
VA conduction to capture the atrium and initiate PMT.
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Sinus tachycardia with pacemaker Wenckebach.
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Safety pacing triggered by timely PVCs. Noted with short AV delays.
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Managed ventricular pacing: AAI mode switches to DDD with short AV delay after
AP without native ventricular beat.
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Sinus tachycardia above upper rate limit; hence no biventricular pacing.
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The atrial lead has dislodged into the ventricle. Ventricular captures are seen
by the first spike (from atrial lead) followed by a functional non-capture from the second spike (from
ventricular lead).
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Normal CRT function with VV delay of 40ms.
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Pacemaker failure. Radiation may cause deterioration of lead and pacemaker
circuit. ECG show asynchronous pacing. Urgent generator change is needed.